Skip to main content

Strain VS Sprain

Strain vs Sprain

STRAINS


s"T"rain = "T"endon


Overstretch injury to musculotendinous unit (tendon)

Concentric: Mxl contraction as the origin and insertion of the mxl comes closer together, Mxl fibres shorten

Eccentric:Origin and insertion move farther apart, the mxl fibres lengthen

Eccentric contraction can produce. greater force within the mxl than concentric contraction, predisposing the mxl to injury at this time


Hypovascular nature of tendons contributes to decreased tissue health, allowing the tendon itself to rupture


Fun fact!

The younger people where the epiphyseal plate in the bone has not yet ossified, the mxl and tendons are stronger than the bone!

Because tendons are moderately vascularized, they are prone to partial or complete rupture at the area of least blood supply either in the middle of the tendon or at the musculotendinous junction.


What is the cause of STRAINS?

Sudden overstitching of the mxl
Extreme contraction of the mxl against heavy resistance

3 grades of Severity

Grade 1: Mild
Minor stretch and/or tear
Minimal loss of strength

Grade 2: Moderate
Tearing of musculotendinous fibres
There may be snapping sensation or sound at time off injury
Unable to continue activity d/t mxl weakness and pain

Grade 3: Severe
Complete rupture of the musculotehdinous unit or an avulsion fracture as the body attachment of the tendon is torn off while the unite remains intact
Palpable/visible gap appears at the injury site
Often mxl shortens and bunches up
Unable to continue activity d/t signifiant pain and mxl weakness

SXS


Acute:

Grade1: Local edema, heat, bruising 

tenderness at site of injury

Little to no loss of strength or ROM

Capable of continuing the activity


Grade2: Tearing of several or many fibres of the musculotendinous unit

Snapping noise or sensation at the site

Gap may be palpable

Moderate tenderness

Moderate pain with activities that contrast or stretch the musculotendinous unit

Moderate loss of strength and ROM
Bruising: Red, black, blue
Hematoma on site


Grade3: Complete rupture
Snapping noise or sensation at the time or injury
Local edema
 Gap palpated in the tissue
Mxl will likely bunch up d/t spasmodic contractions
Severe pain
Immediate loss of strength and ROM
Unable to continue activity
Bruising: Red, black, blue
Hematoma on site
Ruptured mxl in lower limbs is usually surgically repaired and then immobilized in a cast for 4-8wks

Early subacute
Grade1: Little to no pain and reduced strength

Grade2: Pain and moderately reduced strength


Grade3: Pain and markedly reduced strength w/ARROM

Pain, edema and inflammation are still present
Adhesions are developing around the site of injury
b/o poor vasculature, healing is relatively slow
Mxl guarding spasm diminishes
TrP occurs in the affected mxl, its synergist and its antagonists
Reduced ROM

Late subacute
Bruising: Yellow, Green, Brown
Hematoma diminishes
Gap is still palpable
Protective mxl spasm is replaced by increased tone in the affected mxl, its synergists and its antagonists.
TrP occur in affected mxl and in compensatory mxl
Adhesions are maturing around the injury
Reduced ROM

Chronic
No more bruising
Hypertonicity and TrP are present
Adhesions have matured around the injury
Tissue may be cool d/t ischemia
Full ROM of the joint may be reduced with Grade 2 & 3
Repetitive injury from overuse is too stressful for the mxl
Pocket of chronic edema may remain local to the site
Reduced strength of the affected musculotendinous unit and possible disuse atrophy may present

Questions to ask

Health Hx
Rx
Hx of strain (Is this the first time to strain? You have had it before at the same site?)
Did you hear any popping sound when you injured?
Were you assessed by MD?
Do you see anybody else? Chiro, Physio, etc.
Any hematoma, nerve damage or avulsion at the tendon's attachment?
What are you currently feeling?
Palliative & provocative
Swelling on site or distal to the site of injury?
With strain of wt bearing limb, did the limb "Give way" at the time of injury? >>> Indication of Grade 3 strain

Observation

Acute: Gait analysis
Taping or bandages or splint
Edema
Some redness

Early and late subacute:
Bruising
Early subacute-Purple and black
Late subacute-Yellow and green

Chronic:Habituated gait and posture may be observed w/ strain
Visible gap at the lesion site might be present from grade 3 strain
Scar might be formed if musculotendinous unit is reduced surgically

Palpation

Acute
Heat and tenderness present
Firm edema
Palpable gap
Protective mxl spasm
Mxl guarding

Early and Late subacute
Temperature on the site is diminishing
Local tenderness may present
Palpable gap or alteration in the Mxl's contour and hypertonicity 
TrP in affected Mxl

Chronic
Cool to touch d/t ischemia
Tenderness to the lesion site
If it is on shoulder, they may not be able to sleep on affected side
Palpable gap may present w/ Grade 2 & 3
Hypertonicity and TrP may be present
Disuse atrophy in affected mxl

Testing

Acute: ARROM of affected mxl is reduced
Other testing is CONTRAINDICATED in acute stage if Grade2&3 are suspected


How can you tell?

Acute
Snapping sensation at the time of injury?
Difficulty continuing the activity or unable to continue ADL?
Palpa ble gap?
ARROM?

Early and Late Subacute
AROM?
ARROM?
AR isometric contraction?
Grade1: minor loss of strength, minor pain
Grade2: Moderate loss of strength, Pain
Grade3: Significant loss of strength, Pain

Chronic
AROM:May be limited
ARROM
Mxl strength testing: Decreased w/disuse atrophy or Grade 3 strains

Special test for Strain
Thompson's test
Yergason's test(if strain is in the shoulder)
Drop arm test (if strain is in the shoulder)
Aply's scratch test
Thomas and Ely's test
Mxl length test of injured site


Treatment plan

Acute: Mxl is treated with RICE
Positioning: elevate affected area with pillow
Hydrotherapy: Cold

Early & Late Subacute
Stroking and squeezing tech distal to the site of injury
Hydrotherapy: Cold/Warm contrast
TrP
Promote ROM
Lymphatic drainage
Chronic


Outcomes

Acute: Reduce inflammation & edema
Reduce pain
NO direct tx on site

Early & Late subacute: Reduce inflammation & edema
Mid range PROM
Reduce spasm
TrP
Reduce adhesion

Chronic: Reduce sympathetic nervous sys tem firing
Reduce any chronic edema
TrP
Decrease tone
Restore ROM
Promote blood circulation to the injured site
Treat scar if mxl was surgically repaired 

Self Care

Hydrotherapy
Self massage
Maintain strength of the affected mxl in the pain-free manner
Stretch shortened mxl 
Increase strength gradually
Encourage activity

Frequency

Acute: Shorter more frequent tx 
Chronic: Longer, weekly tx
Grade1:Possible to return to activity with a support after 2 days
Grade2:Possible to return to activity after several days to several weeks
Grade3:Immobilization could be removed after 4-8 weeks
Return to activity after 8 weeks but may delay for up to several weeks d/t atrophy of the mxl

SPRAINS

Overstretch injury to a ligament
Trauma related sudden twist or wrench of the joint beyond its normal ROM

Cause

Congenital ligamentous laxity (Hypermobility)
Hx of previous sprains to that joint
Connective tissue pathology such as RA

Grade1-Mild: Minor stretch and tear to the ligament
They can still continue some activities with some discomfort

Grade2-Moderate:Tearing of Lig. fibres occurs
Snapping sound at the time of injury and Jt. gives way
Hyermobile yet stable on passive relaxed testing
They may have difficulty continuing the activity

Grade3-Severe:Complete rupture of the Lig. itself or avulsion fracture as the body attachment of the Lig. is turnoff while the Lig. remains intact
Surgically repaired or treated by the medically conservative approach of immobilization of the Jt. in a cast or strapping

Common sprains
Lateral ankle Lig.
Lateral collateral ligament of ankle joint - Wikipedia

MC Lig. of sprain (Inversion sprain) are 
Ant. talofibular Lig.
Post. talofibular Lig. 
Calcaneofigular Lig. 


Knee Lig.

Knee Ligament Images, Stock Photos & Vectors | Shutterstock
MC Lig. of sprain
MCL, LCL, ACL, PCL,

Wrist Lig.

How to Treat a Sprained Wrist | Sprain, Wrist anatomy, Rotator cuff

MC Lig. to be injured is Scapholunate ligament
Hyperextension of the wrist is usually the cause of the wrist sprain

Shoulder Lig.

Shoulder Injuries Treatment in NJ | Pain Management Doctor, Specialist
Grade1: Acromioclaviular sprain involves tearing of Jt capsule
Grade2:Tear of the Jt capsule and acromioclavidular Lig
Grade3:Tear of the Jt capsule:Acromioclavidular Lig and conoid and 
trapezoid Lig
Fx may also be present

SXS

Acute
Grade1:Minor stretch to Lig
Mild pain, minimal edema, stable Jt
May continue activity

Grade2:Tearing of some or many fibres of the Lig
Snapping noise and Jt gives way
Moderate pain, edema, heat and bruising are present
Slight Jt instability
May have difficulty continuing the activity d/t pain

Grade3:Complete rupture or Avulsion Fx of Lig attachment
Snapping noise, Intense pain, significant edema, heat, bruising
Hematoma, Jt effusion may be present
Jt instability
Unable to continue activity

Early subacute
Grade1:Stable
Grade2:Hypermobile yet stable
Grade3:Hypermobile and unstable w/Liamentous stress testing

Bruising: Black and blue
Pain, edema inflammation are still present but reduced
Adhesions are developing around the injury
d/t hypo vascular characteristic, it heals relatively slow
When protective spasm diminishes, TrP occurs on the site of injury and compensatory mxl
Reduced ROM
Loss of Proprioception at the Jt

Late subacute
Bruising: Yellow, green brown
Pain, edema, inflammation are diminishing
Adhesions are maturing around the injury
Increased tone of mxl crossing the Jt
Affected Jt may still be supported
Reduced ROM
Loss of proprioception at the Jt

Proprioception (or kinesthesia) is the sense though which we perceive the position and movement of our body, including our sense of equilibrium and balance, senses that depend on the notion of force (Jones, 2000)

Chronic
Pain local to the area only if the Lig is stretched
Bruising is gone
Adhesions have matured around the injury
Hyper tonicity and TrP are present
Full ROM of the Jt is restricted
Pocket of Chronic edema may remain local to the Lig
Mxl weakness or disuse atrophy may be present
They may still need some taping to support the Jt

Questions to ask

Health Hx
Any pathology?
Did you hear any snapping sound at the time of injury?
Nerve damage?
Fracture?
Palliative/Provocative


Observation

Acute: Antalgic gait if sprain is in a Wt.bearing Jt
Edema present at the affected Jt
If its Grade3, there may be distal edema present

Early and late subacute
Antalgic gait still present if sprain is in a Wt.bearing Jt
Edema diminishes both on site and distally
Bruising: changes from Purple and black to brown, yellow and green then disappears
If surgically reduced, scars are present

Chronic
Habituated antalgic gait and posture may be observed w/sprain or Wt. bearing Jt
Check Postural assessment!
There may be some edema local to the Lig. usually repetitive sprains of the same Jt
If surgically reduced, scars are present

Palpation

Acute: Hot to touch
Tenderness to the lesion site
Edema is firm
Protective Mxl spasm

Early and late subacute: Heat diminishes as time goes
Tenderness to the lesion site
Edema is less firm and adhesions are resent as healing process from the early to late subacute
Mxl tone becomes tighter and high in late subacute
TrP are present in these Mxl

Chronic
Cool to touch d/t ischemia
Point tenderness occurs locally
Chronic edema:Boggy, jelly-like feeling
Adhesions local to the Lig
Crepitus may present
Hyper tonicity and TrP are local to the site of injury
Reduced ROM

Testing

Acute: ROM of proximal affected and distal jt
Special test: Ballottable patella, Minor effusion test

Early and late subacute:
ROM, RROM
Isometric testing: Mxl crossing the affected jt are strong and painless w/strictly ligamentous injury. If mxl or tendons are also involved, there is pain at the lesion site in the contractile tissue
Special test: Ligamentous stress test, Ant drawer test (Ankle), Valgus or Varus, apple's distraction test, Brush test (Knee), 

Chronic:ROM, RROM
Special test: Ligamentous stress test


Treatment Plan

Acute:RICE
Elevate the injured site
Cold hydrotherapy
Reduce pain and edema
Maintain local circulation proximal to the injury ONLY
Maintain ROM w/ mid range PROM

Early and late subacute
Reduce inflammation, Edema
Prevent from excess adhesion formation
Increase drainage and venous return w/ effleurage, petrissage (Palm kneading, c-scooping, fingertip kneading, ONI tech)
GTO is als used to that jt
Reduce TrP
Mid-range PROM to maintain ROM
Hydrotherapy: Hot/Cold

How do you reduce adhesions?
Short cross-fibre strokes and frictions to the ligament


Chronic
Hydrotherapy:contrast
PROM
Reduce adhesion: Cross fibre, Joint play(gentle)

Contraindications

Distal circulation techniques in acute and early subacute stage because this could increase congestion through the injury site
Friction tech if they are taking anti-inflammatory or blood thinner

Self care for all phase of healing

Hydrotherapy
Self massage
Isometric contraction to strengthen the mxl
ROM to maintain Jt integrity

Treatment frequency

Acute: Short duration & frequent tx
Chronic: Long duration & weekly tx

When can they return to their activity?Grade1: 4-5 days
Grade2:7-14 days
Grade3:6-8 weeks

However...
sprained Lig may take up to 6 months for full maturation of the collagen fibres

It is recommended to receive massage therapy for weekly or biweekly up to 6 months.

Comments

Popular posts from this blog

Tendinitis

Tendinitis Inflammation of a tendon or tendons NOTE : What is the difference between "Tendinitis" and "Tendinosis"? The main difference between  tendinosis  and  tendinitis  is time.  Tendinosis  is a chronic (persistent or recurring) condition caused by repetitive trauma or an injury that hasn't healed. By contrast,  tendinitis  is an acute (sudden, short-term) condition in which inflammation is caused by a direct injury to a tendon (from Google search) Another Link regarding this difference HERE   Okay, From here below, informations are taken from Rattray textbook! Tendon : Made of  regularly arranged, dense collagen fibrils. It appear in 2 shapes: Cord-like structures and broad, sheet-like structures called aponeuroses Limited blood supply compared to Mxl and that makes it slow down of healing process Cause Chronic overload of the tendon leading to micro tearing and inflammatory response in the tendon Cont

Fractures

Break in the continuity of a bone Type of fracture Transverse, oblique, spiral, comminuted, avulsion, osteochondral Common fx names Stage of healing 1. Hematoma within 72H of initial trauma 2.Inflammatory rxn, proliferation of osteoblasts at the periosteum 3.Soft callus or splint is formed from mass of proliferating osteoblast, repair is still incomplete 4.Consolidation occurs as the immature woven bome is changed into mature lamellar bone This is a complete repair 5.Remodeling of the irregular outer surface and reshaping of the marrow space inside the bone take place through alternating osteoclastic and osteoblastic activity Complications of Fx Compartment syndrome This can occur in the forearm and lower legs following fx Nerve compressions This may be indicated by paresthesia in the tissues under the cast Untreated vascular damage It may be indicated by an increase in observable distal red, black or blue bruising Refer to MD!! Bone